Overexpression of Protein Kinase C-η Attenuates Caspase Activation and Tumor Necrosis Factor-α-Induced Cell Death
- 1 December 2000
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 279 (1) , 103-107
- https://doi.org/10.1006/bbrc.2000.3903
Abstract
No abstract availableKeywords
This publication has 27 references indexed in Scilit:
- Proteolytic Cleavage and Activation of Protein Kinase C μ by Caspase-3 in the Apoptotic Response of Cells to 1-β-d-Arabinofuranosylcytosine and Other Genotoxic AgentsJournal of Biological Chemistry, 2000
- Evidence That Protein Kinase Cε Mediates Phorbol Ester Inhibition of Calphostin C- and Tumor Necrosis Factor-α-induced Apoptosis in U937 Histiocytic Lymphoma CellsPublished by Elsevier ,1998
- Caspases: Enemies WithinScience, 1998
- Death Receptors: Signaling and ModulationScience, 1998
- Caspase-3 Is Required for DNA Fragmentation and Morphological Changes Associated with ApoptosisJournal of Biological Chemistry, 1998
- The Proteolytic Cleavage of Protein Kinase C Isotypes, Which Generates Kinase and Regulatory Fragments, Correlates with Fas‐Mediated and 12‐O‐Tetradecanoyl‐Phorbol‐13‐Acetate‐Induced ApoptosisEuropean Journal of Biochemistry, 1997
- TNF-MEDIATED CYTOTOXICITY OF L929 CELLS: ROLE OF STAUROSPORINE IN ENHANCEMENT OF CYTOTOXICITY AND TRANSLOCATION OF PROTEIN KINASE C ISOZYMESCytokine, 1997
- Proteolytic Activation of Protein Kinase C δ by an ICE/CED 3-like Protease Induces Characteristics of ApoptosisThe Journal of Experimental Medicine, 1996
- Contenders in FasL/TNF death signalingCell, 1995
- Intracellular Signaling by Hydrolysis of Phospholipids and Activation of Protein Kinase CScience, 1992