Inflammatory pathogenesis in Alzheimer's disease: biological mechanisms and cognitive sequeli

This publication has 165 references indexed in Scilit:

• α‐Secretase‐Derived Product of β‐Amyloid Precursor Protein Is Decreased by Presenilin 1 Mutations Linked to Familial Alzheimer's Disease
  Journal of Neurochemistry, 1997
• Response of Glial Cells to Ischemia: Roles of Reactive Oxygen Species and Glutathione
  Neuroscience & Biobehavioral Reviews, 1997
• The cognitive effects of recombinant interleukin-2 (rIL-2) therapy: a controlled clinical trial using computerised assessments
  European Journal Of Cancer, 1996
• Cerebrospinal fluid nitrate levels in patients with Alzheimer's disease
  Acta Neurologica Scandinavica, 1996
• RAGE and amyloid-β peptide neurotoxicity in Alzheimer's disease
  Nature, 1996
• Medical hypotheses
  Medical Hypotheses, 1995
• Selective Killing of Cholinergic Neurons by Microglial Activation in Basal Forebrain Mixed Neuronal/Glial Cultures
  Biochemical and Biophysical Research Communications, 1995
• Rodent models of memory dysfunction in Alzheimer's disease and normal aging: Moving beyond the cholinergic hypothesis
  Life Sciences, 1994
• Molecular Mechanisms of Nitric Oxide Actions in the Brain^a
  Annals of the New York Academy of Sciences, 1994
• Microglial cells around amyloid plaques in Alzheimer's disease express leucocyte adhesion molecules of the LFA-1 family
  Neuroscience Letters, 1989