Antisense Gene Therapy for Neurodegenerative Disease?
- 31 March 1997
- journal article
- Published by Elsevier in Experimental Neurology
- Vol. 144 (1) , 139-146
- https://doi.org/10.1006/exnr.1996.6400
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- The antiproliferative activity of c-myb and c-myc antisense oligonucleotides in smooth muscle cells is caused by a nonantisense mechanism.Proceedings of the National Academy of Sciences, 1995
- Genetics and molecular biology of Huntington's diseaseTrends in Neurosciences, 1995
- Long-Term Behavioral Recovery in Parkinsonian Rats by an HSV Vector Expressing Tyrosine HydroxylaseScience, 1994
- Design and application of antisense oligonucleotides in cell culture,in vivo, and as therapeutic agentsCellular and Molecular Neurobiology, 1994
- c-fos antisense generates apomorphine and amphetamine-induced rotationNeuroReport, 1993
- Huntington's disease: animal models and transplantation repairCurrent Opinion in Neurobiology, 1993
- Systemic Administration of a Phosphorothioate Oligonucleotide with a Sequence Complementary to p53 for Acute Myelogenous Leukemia and Myelodysplastic Syndrome: Initial Results of a Phase I TrialAntisense Research and Development, 1993
- Survival and function of intrastriatally grafted primary fibroblasts genetically modified to produce l-dopaNeuron, 1991
- Replication of the neurochemical characteristics of Huntington's disease by quinolinic acidNature, 1986
- Lesion of striatal neurons with kainic acid provides a model for Huntington's choreaNature, 1976