Argyrin A Reveals a Critical Role for the Tumor Suppressor Protein p27kip1 in Mediating Antitumor Activities in Response to Proteasome Inhibition

This publication has 34 references indexed in Scilit:

• Tumor cell-selective regulation of NOXA by c-MYC in response to proteasome inhibition
  Proceedings of the National Academy of Sciences, 2007
• Discovery of an oncogenic activity in p27^Kip1that causes stem cell expansion and a multiple tumor phenotype
  Genes & Development, 2007
• The cell cycle inhibitor p57Kip2 promotes cell death via the mitochondrial apoptotic pathway
  Cell Death & Differentiation, 2007
• p27 Kip1 and Cyclin E Expression and Breast Cancer Survival After Treatment With Adjuvant Chemotherapy
  JNCI Journal of the National Cancer Institute, 2006
• C-terminal phosphorylation controls the stability and function of p27kip1
  The EMBO Journal, 2006
• Testing the importance of p27 degradation by the SCF ^skp2 pathway in murine models of lung and colon cancer
  Proceedings of the National Academy of Sciences, 2006
• Proteasome inhibitors induce a terminal unfolded protein response in multiple myeloma cells
  Blood, 2006
• p27^kip1 independently promotes neuronal differentiation and migration in the cerebral cortex
  Genes & Development, 2006
• Inducible expression of a degradation-resistant form of p27^Kip1 causes growth arrest and apoptosis in breast cancer cells
  FEBS Letters, 2005
• A single cdk inhibitor, p27Xic1, functions beyond cell cycle regulation to promote muscle differentiation inXenopus
  Development, 2003