Chronic exposure of human cells in culture to the tricyclic antidepressant desipramine reduces the number of beta-adrenoceptors
- 1 June 1986
- journal article
- research article
- Published by Elsevier in Biochemical Pharmacology
- Vol. 35 (11) , 1899-1902
- https://doi.org/10.1016/0006-2952(86)90309-6
Abstract
No abstract availableThis publication has 15 references indexed in Scilit:
- Inhibition of phospholipid degradation and changes of the phospholipid-pattern by desipramine in cultured human fibroblastsBiochemical Pharmacology, 1983
- Current concepts on the mechanisms of action of antidepressant drugsPharmacology & Therapeutics, 1981
- Down-regulation of β-adrenergic activity in astroglia by chronic treatment with an antidepressant drugEuropean Journal of Pharmacology, 1981
- Long-Term Antidepressant Treatment Decreases Spiroperidol-Labeled Serotonin Receptor BindingScience, 1980
- ENHANCED ADENOSINE 3': 5' - MONOPHOSPHATE RESPONSE TO BETAADRENERGIC STIMULATION IN CYSTIC FIBROSIS FIBROBLASTS AFTER REMOVAL OF CONDITIONED MEDIUMPediatric Research, 1980
- Effect of electroconvulsive shock on monoaminergic receptor binding sites in rat brainNature, 1979
- Mode of action of antidepressant drugsBiochemical Pharmacology, 1978
- Development of β-adrenergic receptor subsensitivity by antidepressantsNature, 1977
- Inositol phospholipids and cell surface receptor functionBiochimica et Biophysica Acta (BBA) - Reviews on Biomembranes, 1975
- Studies on the Pathogenetic Mechanism of I-Cell Disease in Cultured FibroblastsPediatric Research, 1974