CpG island methylation is a common finding in colorectal cancer cell lines

This publication has 34 references indexed in Scilit:

• Differential involvement of the hypermethylator phenotype in hereditary and sporadic colorectal cancers with high‐frequency microsatellite instability
  Genes, Chromosomes and Cancer, 2001
• Sporadic Colorectal Cancers With Microsatellite Instability and Their Possible Origin in Hyperplastic Polyps and Serrated Adenomas
  JNCI Journal of the National Cancer Institute, 2001
• Kirsten ras mutations in patients with colorectal cancer: the ‘RASCAL II’ study
  British Journal of Cancer, 2001
• Extensive characterization of genetic alterations in a series of human colorectal cancer cell lines
  Oncogene, 2001
• SMAD4 mutations in colorectal cancer probably occur before chromosomal instability, but after divergence of the microsatellite instability pathway
  Proceedings of the National Academy of Sciences, 2001
• Microsatellite instability and the clinicopathological features of sporadic colorectal cancer
  Gut, 2001
• Excessive CpG island hypermethylation in cancer cell lines versus primary human malignancies
  Human Molecular Genetics, 2001
• Methylation of O-6-methylguanine DNA methyltransferase characterizes a subset of colorectal cancer with low-level DNA microsatellite instability.
  2001
• Inactivation of the DNA repair gene O6-methylguanine-DNA methyltransferase by promoter hypermethylation is associated with G to A mutations in K-ras in colorectal tumorigenesis.
  2000
• APC mutations in sporadic colorectal tumors: A mutational “hotspot” and interdependence of the “two hits”
  Proceedings of the National Academy of Sciences, 2000