Prolonged morphine treatment increases rat brain dihydropyridine binding sites: possible involvement in development of morphine dependence
- 1 January 1988
- journal article
- research article
- Published by Elsevier in European Journal of Pharmacology
- Vol. 146 (1) , 73-83
- https://doi.org/10.1016/0014-2999(88)90488-8
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Increased dihydropyridine-sensitive calcium channels in rat brain may underlie ethanol physical dependenceNeuropharmacology, 1987
- Calcium channel inhibitors suppress the morphine‐withdrawal syndrome in ratsBritish Journal of Pharmacology, 1986
- MODIFICATION OF ENDORPHIN/ENKEPHALIN ANALGESIA AND STRESS‐INDUCED ANALGESIA BY DIVALENT CATIONS, A CATION CHELATOR AND AN IONOPHOREBritish Journal of Pharmacology, 1982
- Calcium channels in the somatic membrane of the rat dorsal root ganglion neurons, effect of cAMPBrain Research, 1981
- CLONIDINE BLOCKS ACUTE OPIATE-WITHDRAWAL SYMPTOMSThe Lancet, 1978
- CALCIUM DEPLETION OF SYNAPTOSOMES AFTER MORPHINE TREATMENTBritish Journal of Pharmacology, 1976
- Effects of potassium, veratridine, and scorpion venom on calcium accumulation and transmitter release by nerve terminals in vitro.The Journal of Physiology, 1975
- Development of physical dependence on morphine in respect to time and dosage and quantification of the precipitated withdrawal syndrome in ratsPsychopharmacology, 1973
- Formulation of a Morphine Implantation Pellet Suitable for Tolerance-Physical Dependence Studies in MiceJournal of Pharmaceutical Sciences, 1970
- REGIONAL STUDIES OF CATECHOLAMINES IN THE RAT BRAIN‐IJournal of Neurochemistry, 1966