Immunoadhesins as research tools and therapeutic agents
- 1 April 1997
- journal article
- review article
- Published by Elsevier in Current Opinion in Immunology
- Vol. 9 (2) , 195-200
- https://doi.org/10.1016/s0952-7915(97)80135-5
Abstract
No abstract availableKeywords
This publication has 47 references indexed in Scilit:
- Apo-3, a new member of the tumor necrosis factor receptor family, contains a death domain and activates apoptosis and NF-κBCurrent Biology, 1996
- Deleted in Colorectal Cancer (DCC) Encodes a Netrin ReceptorCell, 1996
- Individually Distinct Ig Homology Domains in PECAM-1 Regulate Homophilic Binding and Modulate Receptor AffinityJournal of Biological Chemistry, 1996
- Inhibition of tumor necrosis factor activity minimizes target organ damage in experimental autoimmune uveoretinitis despite quantitatively normal activated T cell traffic to the retinaEuropean Journal of Immunology, 1996
- SHORT COURSE SINGLE AGENT THERAPY WITH AN LFA-3-IgG1 FUSION PROTEIN PROLONGS PRIMATE CARDIAC ALLOGRAFT SURVIVAL1,2Transplantation, 1996
- CD28/CTLA-4 receptor structure, binding stoichiometry and aggregation during T-cell activationResearch in Immunology, 1995
- Fas(CD95)/FasL interactions required for programmed cell death after T-cell activationNature, 1995
- Cell-autonomous Fas (CD95)/Fas-ligand interaction mediates activation-induced apoptosis in T-cell hybridomasNature, 1995
- Autocrine T-cell suicide mediated by APO-1/(Fas/CD95)Nature, 1995
- A Lymphotoxin-β-Specific ReceptorScience, 1994