Evidence that the ability of imidazoline compounds to stimulate insulin secretion is not due to interaction with σ receptors

This publication has 10 references indexed in Scilit:

• Imidazoline compounds stimulate insulin release by inhibition of K(ATP) channels and interaction with the exocytotic machinery
  Diabetes, 1996
• Imidazoli(di)ne compounds interact with the phencyclidine site of NMDA receptors in the rat brain
  European Journal of Pharmacology, 1996
• Inhibition of 5-HT3 receptor function by imidazolines in mouse neuroblastoma cells: potential involvement of ?2 binding sites
  Naunyn-Schmiedebergs Archiv für experimentelle Pathologie und Pharmakologie, 1996
• Clotrimazole and efaroxan inhibit red cell Gardos channel independently of imidazoline I1 and I2 binding sites
  European Journal of Pharmacology, 1996
• Characterization of the Imidazoline Binding Site Involved in Regulation of Insulin Secretion^fn1
  Annals of the New York Academy of Sciences, 1995
• Clonidine and Cirazoline Inhibit Activation of Nicotinic Channels in PC‐12 Cells^fn1
  Annals of the New York Academy of Sciences, 1995
• Evidence for two different imidazoline sites on pancreatic B cells and vascular bed in rat
  European Journal of Pharmacology, 1995
• Anaesthetic phencyclidine, blocker of the ATP‐sensitive potassium channels
  FEBS Letters, 1994
• Antagonism of the stimulatory effects of efaroxan and glibenclamide in rat pancreatic islets by the imidazoline, RX801080
  British Journal of Pharmacology, 1993
• Stimulation of insulin secretion by imidazoline compounds is not due to interaction with non‐adrenoceptor idazoxan binding sites
  British Journal of Pharmacology, 1993