Urinary excretion of dna repair products correlates with metabolic rates as well as with maximum life spans of different mammalian species
- 1 November 2004
- journal article
- research article
- Published by Elsevier in Free Radical Biology & Medicine
- Vol. 37 (9) , 1449-1454
- https://doi.org/10.1016/j.freeradbiomed.2004.07.014
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- Oxidative DNA damage in cancer patients: a cause or a consequence of the disease development?Mutation Research - Fundamental and Molecular Mechanisms of Mutagenesis, 2003
- Oxidative DNA damage: assessment of the role in carcinogenesis, atherosclerosis, and acquired immunodeficiency syndrome1 1This article is part of a series of reviews on “Oxidative DNA Damage and Repair.” The full list of papers may be found on the homepage of the journal.Free Radical Biology & Medicine, 2002
- Genomic Priorities in AgingScience, 2002
- Oxidative damage to mitochondrial DNA is inversely related to maximum life span in the heart and brain of mammalsThe FASEB Journal, 2000
- Oxidative DNA damage processing in nuclear and mitochondrial DNABiochimie, 1999
- The Free Radical Theory of Aging MaturesPhysiological Reviews, 1998
- Aging processes, DNA damage, and repair 1The FASEB Journal, 1997
- Antioxidants and agingThe American Journal of Clinical Nutrition, 1991
- Oxidative damage to DNA: relation to species metabolic rate and life span.Proceedings of the National Academy of Sciences, 1988
- Aging: A Theory Based on Free Radical and Radiation ChemistryJournal of Gerontology, 1956