Methylation of CpG island is not a ubiquitous mechanism for the loss of oestrogen receptor in breast cancer cells.
Open Access
- 1 January 1998
- journal article
- research article
- Published by Springer Nature in British Journal of Cancer
- Vol. 77 (2) , 181-185
- https://doi.org/10.1038/bjc.1998.31
Abstract
No abstract availableKeywords
This publication has 24 references indexed in Scilit:
- Irreversible loss of the oestrogen receptor in T47D breast cancer cells following prolonged oestrogen deprivationBritish Journal of Cancer, 1996
- Silencing of the VHL tumor-suppressor gene by DNA methylation in renal carcinoma.Proceedings of the National Academy of Sciences, 1994
- Methylation of the oestrogen receptor CpG island links ageing and neoplasia in human colonNature Genetics, 1994
- Methylation of the estrogen receptor gene CpG island marks loss of estrogen receptor expression in human breast cancer cells.1994
- DNA methylation and genomic imprintingCell, 1994
- Deletions of the cyclin-dependent kinase-4 inhibitor gene in multiple human cancersNature, 1994
- Growth Regulation of Estrogen Receptor-Negative Breast Cancer Cells Transfected With Complementary DNAs for Estrogen ReceptorJNCI Journal of the National Cancer Institute, 1992
- CHARACTERIZATION OF A RECEPTOR-NEGATIVE, HORMONE-NONRESPONSIVE CLONE DERIVED FROM A T47D HUMAN BREAST-CANCER CELL-LINE KEPT UNDER ESTROGEN-FREE CONDITIONS1990
- Allelic loss of chromosomes 16q and 10q in human prostate cancer.Proceedings of the National Academy of Sciences, 1990
- Variability of DNA methylation patterns during serial passage of human diploid fibroblastsProceedings of the National Academy of Sciences, 1982