Osmotic Diuresis Due to Retained Urea after Release of Obstructive Uropathy

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Abstract
POLYURIA after the release of obstructive uropathy is a clinical entity of considerable medical interest. The prompt restoration of effective glomerular filtration may be associated with a delayed recovery of tubular function resulting in exaggerated polyuria. Both salt-losing nephritis and nephrogenic diabetes insipidus have been described.1 , 2 We have encountered a clinical case of massive diuresis that followed release of urinary-tract obstruction in which urea was proved to be the osmotic driving force. The observation is noteworthy because identification of the type of diuresis carries specific therapeutic implications.Case ReportAn 80-year-old man was admitted to the hospital because of syncope. . . .