Novel bile acid derivatives induce apoptosis via a p53-independent pathway in human breast carcinoma cells
- 9 January 2001
- journal article
- research article
- Published by Elsevier in Cancer Letters
- Vol. 163 (1) , 83-93
- https://doi.org/10.1016/s0304-3835(00)00671-6
Abstract
No abstract availableKeywords
This publication has 37 references indexed in Scilit:
- A novel role for ursodeoxycholic acid in inhibiting apoptosis by modulating mitochondrial membrane perturbation.Journal of Clinical Investigation, 1998
- Different bile acids exhibit distinct biological effects: The tumor promoter deoxycholic acid induces apoptosis and the chemopreventive agent ursodeoxycholic acid inhibits cell proliferationNutrition and Cancer, 1998
- The Baxα:Bcl-2 ratio modulates the response to dexamethasone in leukaemic cells and is highly variable in childhood acute leukaemiaInternational Journal of Cancer, 1997
- Cyclin‐dependent protein kinases: Key regulators of the eukaryotic cell cycleBioEssays, 1995
- Inhibitors of mammalian G1 cyclin-dependent kinases.Genes & Development, 1995
- The retinoblastoma protein and cell cycle controlCell, 1995
- Chromatin condensation during apoptosis is accompanied by degradation of lamin A+B, without enhanced activation of cdc2 kinase.The Journal of cell biology, 1994
- WAF1, a potential mediator of p53 tumor suppressionCell, 1993
- Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programed cell deathCell, 1993
- FÆCAL BILE-ACIDS AND CLOSTRIDIA IN PATIENTS WITH CANCER OF THE LARGE BOWELThe Lancet, 1975