Aberrant Wound Healing and TGF-β Production in the Autoimmune-Prone MRL/+ Mouse
- 1 September 1999
- journal article
- Published by Elsevier in Clinical Immunology
- Vol. 92 (3) , 300-310
- https://doi.org/10.1006/clim.1999.4754
Abstract
No abstract availableKeywords
This publication has 60 references indexed in Scilit:
- Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-beta, PGE2, and PAF.Journal of Clinical Investigation, 1998
- Effect of Age and Caloric Restriction on Cutaneous Wound Closure in Rats and MonkeysThe Journals of Gerontology: Series A, 1997
- Apoptosis by Death FactorCell, 1997
- Differential effects of transforming growth factor‐β1 on IgA vs. IgG2b production by lipopolysaccharide‐stimulated lymph node B cells: a comparative study with spleen B cellsEuropean Journal of Immunology, 1996
- Spontaneous elaboration of transforming growth factor beta suppresses host defense against bacterial infection in autoimmune MRL/lpr mice.The Journal of Experimental Medicine, 1994
- Transforming growth factor-β1 enhances IgG and IgA sheep red blood cell responsesInternational Immunology, 1992
- Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosisNature, 1992
- Immunoregulatory effects of transforming growth factor-β in a prolonged period of cultureCellular Immunology, 1992
- The Transforming Growth Factor-beta FamilyAnnual Review of Cell Biology, 1990
- Tumors: Wounds That Do Not HealNew England Journal of Medicine, 1986