Myc Antagonizes Ras-mediated Growth Arrest in Leukemia Cells through the Inhibition of the Ras-ERK-p21Cip1 Pathway

This publication has 85 references indexed in Scilit:

• Subcellular Localization Determines the Protective Effects of Activated ERK2 against Distinct Apoptogenic Stimuli in Myeloid Leukemia Cells
  Journal of Biological Chemistry, 2004
• Activation of H-Ras in the Endoplasmic Reticulum by the RasGRF Family Guanine Nucleotide Exchange Factors
  Molecular and Cellular Biology, 2004
• Abolition of Cyclin-Dependent Kinase Inhibitor p16^Ink4a and p21^Cip1/Waf1 Functions Permits Ras-Induced Anchorage-Independent Growth in Telomerase-Immortalized Human Fibroblasts
  Molecular and Cellular Biology, 2003
• Differences on the Inhibitory Specificities of H-Ras, K-Ras, and N-Ras (N17) Dominant Negative Mutants Are Related to Their Membrane Microlocalization
  Journal of Biological Chemistry, 2003
• Myc suppression of the p21Cip1 Cdk inhibitor influences the outcome of the p53 response to DNA damage
  Nature, 2002
• E7 Abolishes Raf-Induced Arrest via Mislocalization of p21^Cip1
  Molecular and Cellular Biology, 2002
• Nerve Growth Factor Uses Ras/ERK and Phosphatidylinositol 3-Kinase Cascades to Up-regulate theN-Methyl-d-aspartate Receptor 1 Promoter
  Journal of Biological Chemistry, 2001
• Malignant transformation by cyclin E and Ha-Ras correlates with lower sensitivity towards induction of cell death but requires functional Myc and CDK4
  Oncogene, 1997
• Max and inhibitory c-Myc mutants induce erythroid differentiation and resistance to apoptosis in human myeloid leukemia cells
  Oncogene, 1997
• Proto-oncogene expression in differentiating and non-differentiating chronic myelogenous leukaemia cells
  European Journal of Cancer and Clinical Oncology, 1990