Systemic 3-nitropropionic acid: long-term effects on locomotor behavior
- 1 May 1994
- journal article
- research article
- Published by Elsevier in Brain Research
- Vol. 646 (2) , 242-246
- https://doi.org/10.1016/0006-8993(94)90085-x
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- Neurochemical and histologic characterization of striatal excitotoxic lesions produced by the mitochondrial toxin 3-nitropropionic acidJournal of Neuroscience, 1993
- Striatal degeneration induced by mitochondrial blockade is prevented by biologically delivered NGFJournal of Neuroscience Research, 1993
- Age‐Dependent Vulnerability of the Striatum to the Mitochondrial Toxin 3‐Nitropropionic AcidJournal of Neurochemistry, 1993
- Age dependence of striatal neuronal death caused by mitochondrial dysfunctionNeuroReport, 1993
- Does impairment of energy metabolism result in excitotoxic neuronal death in neurodegenerative illnesses?Annals of Neurology, 1992
- Nigral dopamine type-1 receptors are reduced in Huntington's disease: A postmortem autoradiographic study using [3H]SCH 23390 and correlation with [3H]forskolin bindingExperimental Neurology, 1990
- Excitatory amino acid neurotoxicity at the N-methyl-d-aspartate receptor in cultured neurons: role of the voltage-dependent magnesium blockBrain Research, 1989
- Nature and distribution of brain lesions in rats intoxicated with 3-nitropropionic acid: A type of hypoxic (energy deficient) brain damageActa Neuropathologica, 1987
- Regional Mitochondrial Respiratory Activity in Huntington's Disease BrainJournal of Neurochemistry, 1985
- Lesion of striatal neurons with kainic acid provides a model for Huntington's choreaNature, 1976