DOXORUBICIN-INDUCED SKIN ULCER IN THE PIGLET
- 1 January 1983
- journal article
- research article
- Vol. 67 (12) , 1075-1078
Abstract
Skin ulceration produced by inadvertently extravasated doxorubicin is characterized by a prolonged course accompanied by severe morbidity, and it has proven to be notoriously difficult to treat. In attempts to identify possible antidotes, 11 different pharmacologic agents [carnitine, coenzyme Q10, deferoxamine mesylate, dimethyl sulfoxide (DMSO), dopamine, DNA type V, human serum albumin, iron dextran, isoproterenol, NaHCD3, .alpha.-tocopherol] were tested using piglets, because their skin is anatomically similar to that of man. Among the agents studied, topical application of DMSO daily for 7 days tended to decrease the maximal diameter and accelerate healing of skin ulcers produced by intradermal doxorubicin. .alpha.-Tocopherol appeared to worsen the ulceration. None of the 11 agents studied prevented the development of ulcerations completely.This publication has 11 references indexed in Scilit:
- Treatment of tissue extravasation by antitumor agentsCancer, 1982
- Soft-Tissue Necrosis Induced by Extravasated Cancer Chemotherapeutic Agents2, 3JNCI Journal of the National Cancer Institute, 1981
- PHARMACOLOGIC ANTIDOTES TO EXPERIMENTAL DOXORUBICIN SKIN TOXICITY - A SUGGESTED ROLE FOR BETA-ADRENERGIC COMPOUNDS1981
- Experimental model of doxorubicin extravasation in the mouseJournal of Pharmacological Methods, 1980
- Interaction of human serum albumin with anticancer agents in vitroBritish Journal of Cancer, 1980
- EXPERIMENTAL SKIN NECROSIS PRODUCED BY ADRIAMYCIN1979
- PHASE-I CLINICAL-STUDY OF QUELAMYCIN1978
- Clinical course and management of accidental adriamycin extravasationCancer, 1977
- Skin ulcers due to adriamycinCancer, 1976
- AdriamycinAnnals of Internal Medicine, 1974