Loss-of-Function Mutations in PPARγ Associated with Human Colon Cancer
- 1 June 1999
- journal article
- Published by Elsevier in Molecular Cell
- Vol. 3 (6) , 799-804
- https://doi.org/10.1016/s1097-2765(01)80012-5
Abstract
No abstract availableKeywords
This publication has 25 references indexed in Scilit:
- Activation of PPARγ leads to inhibition of anchorage-independent growth of human colorectal cancer cellsGastroenterology, 1998
- Obesity Associated with a Mutation in a Genetic Regulator of Adipocyte DifferentiationNew England Journal of Medicine, 1998
- PPARγ Promotes Monocyte/Macrophage Differentiation and Uptake of Oxidized LDLCell, 1998
- Oxidized LDL Regulates Macrophage Gene Expression through Ligand Activation of PPARγCell, 1998
- Mutation spectrum and genotype-phenotype analyses in Cowden disease and Bannayan-Zonana syndrome, two hamartoma syndromes with germline PTEN mutationHuman Molecular Genetics, 1998
- Terminal Differentiation of Human Breast Cancer through PPARγMolecular Cell, 1998
- Germline mutations of the PTEN gene in Cowden disease, an inherited breast and thyroid cancer syndromeNature Genetics, 1997
- Inhibition of Adipogenesis Through MAP Kinase-Mediated Phosphorylation of PPARγScience, 1996
- 15-Deoxy-Δ12,14-Prostaglandin J2 is a ligand for the adipocyte determination factor PPARγCell, 1995
- A genetic model for colorectal tumorigenesisCell, 1990