p27Kip 1 Expression by Contact Inhibition as a Prognostic Index of Human Glioma
Open Access
- 1 April 2000
- journal article
- research article
- Published by Wiley in Journal of Neurochemistry
- Vol. 74 (4) , 1393-1399
- https://doi.org/10.1046/j.1471-4159.2000.0741393.x
Abstract
The clinical manifestations of human glioma are known to be diverse, ranging from aggressive growth and invasion to apparent dormancy; however, the molecular mechanism underlying this dive...Keywords
This publication has 22 references indexed in Scilit:
- p27 Modulates Cell Cycle Progression and Chemosensitivity in Human Malignant GliomaBiochemical and Biophysical Research Communications, 1999
- Up‐regulation of p27Kip1 Correlates Inversely with Anchorage‐independent Growth of Human Cancer Cell LinesJapanese Journal of Cancer Research, 1998
- Differences in the mechanisms of growth control in contact-inhibited and serum-deprived human fibroblastsOncogene, 1997
- A recombinant adenovirus expressing p27Kip1 induces cell cycle arrest and loss of cyclin-Cdk activity in human breast cancer cellsOncogene, 1997
- Translational Control of p27 Kip1 Accumulation During the Cell CycleScience, 1996
- Protein expression of the α, γ, δ and ε subspecies of protein kinase C changes as C6 glioma cells become contact inhibited and quiescent in the presence of serumFEBS Letters, 1995
- Cloning of p27Kip1, a cyclin-dependent kinase inhibitor and a potential mediator of extracellular antimitogenic signalsCell, 1994
- p27Kip1, a cyclin-Cdk inhibitor, links transforming growth factor-beta and contact inhibition to cell cycle arrest.Genes & Development, 1994
- Invasiveness of human glioma cell lines in vitro: Relation to tumorigenicity in athymic miceActa Neuropathologica, 1987
- Contact inhibition and malignancyNature, 1979