Critical Role of Smads and AP-1 Complex in Transforming Growth Factor-β-dependent Apoptosis
Open Access
- 1 November 2000
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 275 (46) , 36295-36302
- https://doi.org/10.1074/jbc.m006023200
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- Regulation of Smad ActivityCell, 2000
- TGF-β SIGNAL TRANSDUCTIONAnnual Review of Biochemistry, 1998
- Transforming Growth Factor-β Stimulates Interleukin-11 Transcription via Complex Activating Protein-1-dependent PathwaysJournal of Biological Chemistry, 1998
- Induction of Apoptosis by DPC4, a Transcriptional Factor Regulated by Transforming Growth Factor-β through Stress-activated Protein Kinase/c-Jun N-terminal Kinase (SAPK/JNK) Signaling PathwayPublished by Elsevier ,1997
- Dissection of TNF Receptor 1 Effector Functions: JNK Activation Is Not Linked to Apoptosis While NF-κB Activation Prevents Cell DeathCell, 1996
- Receptor-associated Mad homologues synergize as effectors of the TGF-β responseNature, 1996
- DPC4 , A Candidate Tumor Suppressor Gene at Human Chromosome 18q21.1Science, 1996
- Role of transforming growth factor beta 1 on hepatic regeneration and apoptosis in liver diseases.Journal of Clinical Pathology, 1995
- A c-jun dominant negative mutant protects sympathetic neurons against programmed cell deathNeuron, 1995
- Purified transcription factor AP-1 interacts with TPA-inducible enhancer elementsCell, 1987