Extensive nerve overgrowth and paucity of the tailed asymmetric form (16 S) of acetylcholinesterase in the developing skeletal neuromuscular system of the dysgenic () mouse
- 1 January 1984
- journal article
- research article
- Published by Elsevier in Developmental Biology
- Vol. 101 (1) , 181-191
- https://doi.org/10.1016/0012-1606(84)90128-3
Abstract
No abstract availableThis publication has 32 references indexed in Scilit:
- Crosslinking and binding of fibronectin with asymmetric acetylcholinesteraseBiochemistry, 1981
- Neural influence on acetylcholine receptor clusters in embryonic development of skeletal musclesNature, 1979
- The Dependence of Acetylcholinesterase Aggregation at Low Ionic Strength upon a Polyanionic ComponentEuropean Journal of Biochemistry, 1978
- The distribution of α‐bungarotoxin binding sites on mammalian skeletal muscle developing in vivoThe Journal of Physiology, 1977
- Muscular Dysgenesis in the Mouse (mdg/mdg)Journal of Neuropathology and Experimental Neurology, 1977
- Multiple forms of acetylcholinesterase and their distribution in endplate and non‐endplate regions of rat diaphragm muscleJournal of Neurobiology, 1973
- An in vitro study of normal and mutant myogenesis in the mouseDevelopmental Biology, 1972
- A Modified Pyridine-Silver Stain for Teased Preparations of Motor and Sensory Nerve Endings in Skeletal MuscleStain Technology, 1970
- Lethal Genes and Analysis of DifferentiationScience, 1963
- A new and rapid colorimetric determination of acetylcholinesterase activityBiochemical Pharmacology, 1961