Akt Blocks Breast Cancer Cell Motility and Invasion through the Transcription Factor NFAT
Open Access
- 1 November 2005
- journal article
- Published by Elsevier in Molecular Cell
- Vol. 20 (4) , 539-550
- https://doi.org/10.1016/j.molcel.2005.10.033
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- Lack of PTEN sequesters CHK1 and initiates genetic instabilityCancer Cell, 2005
- Stabilization of Mdm2 via Decreased Ubiquitination Is Mediated by Protein Kinase B/Akt-dependent PhosphorylationJournal of Biological Chemistry, 2004
- Activation of Akt-1 (PKB-α) Can Accelerate ErbB-2-Mediated Mammary Tumorigenesis but Suppresses Tumor InvasionCancer Research, 2004
- Tumour-cell invasion and migration: diversity and escape mechanismsNature Reviews Cancer, 2003
- Akt Activation Promotes Degradation of Tuberin and FOXO3a via the ProteasomeJournal of Biological Chemistry, 2003
- The phosphatidylinositol 3-Kinase–AKT pathway in human cancerNature Reviews Cancer, 2002
- The Phosphoinositide 3-Kinase PathwayScience, 2002
- Akt/PKB promotes cancer cell invasion via increased motility and metalloproteinase productionThe FASEB Journal, 2001
- Inhibition of glycogen synthase kinase-3 by insulin mediated by protein kinase BNature, 1995
- NF-AT components define a family of transcription factors targeted in T-cell activationNature, 1994