Abstract
New developments in the chemotherapy of tuberculosis have thrown present policies and practices regarding isoniazid (INH) chemoprophylaxis into question. The eradication of tuberculosis by the systematic treatment of tuberculin reactors with INH was proposed during the 1950's. After experimental studies had rendered encouraging results in 1959, Lambert1 questioned the role of INH as a chemoprophylactic agent. He asked, "How effective is INH in preventing tuberculous disease? What are the risks of drug toxicity in large populations treated with INH?"The efficacy of INH chemoprophylactic management of tuberculin reactors was well established in large, well controlled and triple-blind studies* conducted by . . .

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