Bax translocation to mitochondria subsequent to a rapid loss of mitochondrial membrane potential
- 28 August 2001
- journal article
- Published by Springer Nature in Cell Death & Differentiation
- Vol. 8 (9) , 909-920
- https://doi.org/10.1038/sj.cdd.4400889
Abstract
Cell death and differentiation is a monthly research journal focused on the exciting field of programmed cell death and apoptosis. It provides a single accessible source of information for both scientists and clinicians, keeping them up-to-date with advances in the field. It encompasses programmed cell death, cell death induced by toxic agents, differentiation and the interrelation of these with cell proliferation.Keywords
This publication has 67 references indexed in Scilit:
- Conformation of the Bax C-terminus regulates subcellular location and cell deathThe EMBO Journal, 1999
- Bax-induced Caspase Activation and Apoptosis via Cytochromec Release from Mitochondria Is Inhibitable by Bcl-xLJournal of Biological Chemistry, 1999
- Enforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosisThe EMBO Journal, 1998
- Widespread Elimination of Naturally Occurring Neuronal Death inBax-Deficient MiceJournal of Neuroscience, 1998
- Movement of Bax from the Cytosol to Mitochondria during ApoptosisThe Journal of cell biology, 1997
- Caspase-dependent apoptosis of COS-7 cells induced by Bax overexpression: differential effects of Bcl-2 and Bcl-xL on Bax-induced caspase activation and apoptosisOncogene, 1997
- Structure-Function Comparisons of the Proapoptotic Protein Bax in Yeast and Mammalian CellsMolecular and Cellular Biology, 1996
- Bax-Deficient Mice with Lymphoid Hyperplasia and Male Germ Cell DeathScience, 1995
- BH1 and BH2 domains of Bcl-2 are required for inhibition of apoptosis and heterodimerization with BaxNature, 1994
- Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programed cell deathCell, 1993