Abstract
THE occurrence of hyperchloremic acidosis after bilateral ureterosigmoidostomy has been reported frequently and has caused much medical interest and many changes in surgical technics.1 2 3 The underlying physiologic features that account for this picture center around two general concepts. The first concerns intestinal absorption of urinary constituents and selective absorption of chloride, sodium and chloride as sodium chloride and ammonium and chloride, and the second abnormalities in function of the renal tubules. According to Lapides2 such abnormalities do not occur if renal function is good. In some cases with presumably normal preoperative renal function hyperchloremic acidosis has developed postoperatively, and a . . .