Heterodimer formation between c-Jun and Jun B proteins mediated by Epstein–Barr virus encoded latent membrane protein 1

This publication has 44 references indexed in Scilit:

• AP-1 complexes containing cJun and JunB cause cellular transformation of Rat1a fibroblasts and share transcriptional targets
  Oncogene, 2003
• Cells in G2/M phase increased in human nasopharyngeal carcinoma cell line by EBV-LMP1 through activation of NF-κB and AP-1
  Cell Research, 2003
• Amplification and overexpression of JUNB is associated with primary cutaneous T-cell lymphomas
  Blood, 2003
• AP-1 as a regulator of cell life and death
  Nature Cell Biology, 2002
• Role of JunB in Erythroid Differentiation
  Journal of Biological Chemistry, 2002
• JunB negatively regulates AP-1 activity and cell proliferation of malignant mouse keratinocytes
  Zeitschrift für Krebsforschung und Klinische Onkologie, 2001
• Loss of JunB Activity Enhances Stromelysin 1 Expression in a Model of the Epithelial-to-Mesenchymal Transition of Mouse Skin Tumors
  Molecular and Cellular Biology, 2001
• cJun overexpression in MCF-7 breast cancer cells produces a tumorigenic, invasive and hormone resistant phenotype
  Oncogene, 1999
• Neoplastic transformation of rat thyroid cells requires the junB and fra-1 gene induction which is dependent on the HMGI-C gene product
  The EMBO Journal, 1997
• Purified transcription factor AP-1 interacts with TPA-inducible enhancer elements
  Cell, 1987