Bax:Bcl-2 ratio modulation by bryostatin 1 and novel antitubulin agents is important for susceptibility to drug induced apoptosis in the human early pre-B acute lymphoblastic leukemia cell line, Reh

This publication has 41 references indexed in Scilit:

• Mechanism of UV-Induced Apoptosis in Human Leukemia Cells: Roles of Ca2+/Mg2+-Dependent Endonuclease, Caspase-3, and Stress-Activated Protein Kinases
  Experimental Cell Research, 1998
• Synergistic interaction of selected marine animal anticancer drugs against human diffuse large cell lymphoma
  Anti-Cancer Drugs, 1998
• The Baxα:Bcl-2 ratio modulates the response to dexamethasone in leukaemic cells and is highly variable in childhood acute leukaemia
  International Journal of Cancer, 1997
• TGF-β inhibits growth and induces apoptosis in leukemic B cell precursors
  Leukemia, 1997
• Localization of Granzyme B in the Nucleus
  Published by Elsevier ,1996
• The bcl-2 and p53 oncoproteins can be modulated by bryostatin 1 and dolastatins in human diffuse large cell lymphoma
  Anti-Cancer Drugs, 1995
• High expression of bcl-2 is the rule in acute lymphoblastic leukemia, except in Burkitt subtype at presentation, and is not correlated with the prognosis
  Annals of Hematology, 1994
• Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programed cell death
  Cell, 1993
• Comparative effects of polymyxin B, phorbol ester and bryostatin on protein phosphorylation, protein kinase C translocation, phospholipid metabolism and differentiation of HL60 cells
  Biochemical and Biophysical Research Communications, 1987
• Surface immunoglobulin light-chain expression by the “common” all cell line REH
  Clinical Immunology and Immunopathology, 1985