The Mortician's Mystery

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Abstract
ALTHOUGH many causes of gynecomastia have been described, all cases of this disorder appear to share the essential pathophysiologic feature of a relative increase in estrogenic activity, whether endogenous or exogenous, a decrease in androgenic activity, or both.1 , 2 Like gynecomastia, hypogonadotropic hypogonadism in men has been reported to have many sources, most of which are mass lesions or infiltrative disorders of the pituitary gland or hypothalamus, idiopathic panhypopituitarism, or isolated gonadotropin-releasing hormone deficiency.3 4 5 Although hypogonadotropic hypogonadism frequently is reversible in women,4 , 6 it rarely is in men except in patients who have prolactin-secreting or estrogen-secreting tumors or hemochromatosis, or who have . . .