Hepatocyte Resistance to Oxidative Stress Is Dependent on Protein Kinase C-mediated Down-regulation of c-Jun/AP-1
Open Access
- 1 July 2004
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 279 (30) , 31089-31097
- https://doi.org/10.1074/jbc.m404170200
Abstract
No abstract availableKeywords
This publication has 43 references indexed in Scilit:
- Chelerythrine is a potent and specific inhibitor of protein kinase CPublished by Elsevier ,2004
- Protein Kinase Cδ Selectively Regulates Protein Kinase D-Dependent Activation of NF-κB in Oxidative Stress SignalingMolecular and Cellular Biology, 2004
- Vasopressin‐induced intracellular redistribution of protein kinase D in intestinal epithelial cellsJournal of Cellular Physiology, 2003
- AP-1 as a regulator of cell life and deathNature Cell Biology, 2002
- Protein kinase D complexes with C-Jun N-terminal kinase via activation loop phosphorylation and phosphorylates the C-Jun N-terminusOncogene, 2002
- Activation Loop Ser744 and Ser748 in Protein Kinase D Are Transphosphorylated in VivoJournal of Biological Chemistry, 2001
- Protein Kinase D Is Sufficient to Suppress EGF-Induced c-Jun Ser 63 PhosphorylationBiochemical and Biophysical Research Communications, 2001
- Oxidative Stress Induces Protein Kinase D Activation in Intact CellsJournal of Biological Chemistry, 2000
- NFkappaB prevents apoptosis and liver dysfunction during liver regeneration.Journal of Clinical Investigation, 1998
- Secretion, Surface Localization, Turnover, and Steady State Expression of Protein Disulfide Isomerase in Rat HepatocytesPublished by Elsevier ,1995