Elevation of neuronal MAO-B activity in a transgenic mouse model does not increase sensitivity to the neurotoxin 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)
- 5 September 1994
- journal article
- Published by Elsevier in Brain Research
- Vol. 656 (1) , 108-114
- https://doi.org/10.1016/0006-8993(94)91371-4
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- Effect of Deprenyl on the Progression of Disability in Early Parkinson's DiseaseNew England Journal of Medicine, 1989
- MPTP treatment combined with ethanol or acetaldehyde selectively destroys dopaminergic neurons in mouse substantia nigraBrain Research, 1989
- GM1 ganglioside treatment promotes recovery of striatal dopamine concentrations in the mouse model of MPTP-induced ParkinsonismExperimental Neurology, 1989
- A high dose of MPTP overcomes the protective effect of selegiline against dopaminergic neurotoxicityJournal of Pharmacy and Pharmacology, 1989
- Differences between monoamine oxidase concentrations in striatum and forebrain of aged and young ratsJournal Of Neural Transmission-Parkinsons Disease and Dementia Section, 1988
- Strain differences in systematic 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine neurotoxicity in mice correlate best with monoamine oxidase activity at the blood-brain barrierLife Sciences, 1988
- EC array sensor concepts and dataLife Sciences, 1987
- Difference in monoamine oxidase B activity between C57 black and albino NMRI mouse strains may explain differential effects of the neurotoxin MPTPNeuroscience Letters, 1987
- Protection against the dopaminergic neurotoxicity of 1-methyl-4-phenyl-1,2,5,6-tetrahydropyridine by monoamine oxidase inhibitorsNature, 1984
- The effect of age on the activity and molecular properties of human brain monoamine oxidaseJournal Of Neural Transmission-Parkinsons Disease and Dementia Section, 1980