No genetic association between polymorphisms in the Fyn kinase gene and age of schizophrenic onset
- 12 April 2003
- journal article
- research article
- Published by Elsevier in Neuroscience Letters
- Vol. 343 (1) , 70-72
- https://doi.org/10.1016/s0304-3940(03)00310-0
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Culture, Sports, Science and Technology (11770562)
This publication has 14 references indexed in Scilit:
- Expression of Fyn, a non-receptor tyrosine kinase in prefrontal cortex from patients with schizophrenia and its correlation with clinical onsetMolecular Brain Research, 2003
- Overexpression of Fyn tyrosine kinase causes abnormal development of primary sensory neurons in Xenopus laevis embryosDevelopment, Growth & Differentiation, 2001
- Neurodevelopmental hypothesis of schizophrenia: a central sensory disturbanceMedical Hypotheses, 2000
- Follow-up study on a susceptibility locus for schizophrenia on chromosome 6qAmerican Journal of Medical Genetics, 1999
- Suggestive Evidence for a Schizophrenia Susceptibility Locus on Chromosome 6q and a Confirmation in an Independent Series of PedigreesGenomics, 1997
- Schizophrenia: a disorder of neurodevelopment?Current Opinion in Neurobiology, 1997
- Susceptibility to drug-induced seizures of Fyn tyrosine kinase-deficient miceNeuroReport, 1996
- Initial events of myelination involve Fyn tyrosine kinase signallingNature, 1994
- The protein tyrosine kinase, fyn, in Alzheimerʼs disease pathologyNeuroReport, 1993
- Stimulation of Protein Tyrosine Phosphorylation by NMDA Receptor ActivationScience, 1991