Preferential Loss of Fhit Expression in Signet-Ring Cell and Krukenberg Subtypes of Gastric Cancer
Open Access
- 1 September 2002
- journal article
- research article
- Published by Elsevier in Laboratory Investigation
- Vol. 82 (9) , 1201-1208
- https://doi.org/10.1097/01.lab.0000028060.37763.d5
Abstract
No abstract availableKeywords
This publication has 26 references indexed in Scilit:
- Correlation of histologic subtypes and replication error phenotype with comparative genomic hybridization in gastric cancerGenes, Chromosomes and Cancer, 2000
- Molecular Bases of Human Stomach CarcinogenesisPublished by Springer Nature ,1997
- The FHIT Gene at 3p14.2 Is Abnormal in Lung CancerCell, 1996
- Fhit, a Putative Tumor Suppressor in Humans, Is a Dinucleoside 5‘,5‘ ‘‘-P1,P3-Triphosphate HydrolaseBiochemistry, 1996
- Gastric CarcinomaNew England Journal of Medicine, 1995
- Molecular mechanism of stomach carcinogenesisZeitschrift für Krebsforschung und Klinische Onkologie, 1993
- Incidence and prognosis of Krukenberg tumour in Northern IrelandBritish Journal of Surgery, 1992
- Cytogenetic analysis of a cell line established from a Krukenberg tumorCancer Genetics and Cytogenetics, 1990
- Characterization of a continuous cell line in culture established from a Krukenberg tumour of the ovary arising from a primary gastric adenocarcinomaEuropean Journal of Cancer and Clinical Oncology, 1988
- KRUKENBERGʼS TUMORS. A SURVEY OF FORTY-FOUR CASESAnnals of Surgery, 1942