Overexpression of p27Kip1 induces growth arrest and apoptosis in an oral cancer cell line
- 1 October 2002
- journal article
- Published by Elsevier in Oral Oncology
- Vol. 38 (7) , 730-736
- https://doi.org/10.1016/s1368-8375(02)00011-8
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Culture, Sports, Science and Technology
This publication has 21 references indexed in Scilit:
- Prognostic Significance of p27 Expression in Carcinoma of the Oral Cavity and OropharynxThe Laryngoscope, 1999
- A recombinant adenovirus expressing p27Kip1 induces cell cycle arrest and loss of cyclin-Cdk activity in human breast cancer cellsOncogene, 1997
- Crystal structure of the p27Kip1 cyclin-dependent-kinase inibitor bound to the cyclin A–Cdk2 complexNature, 1996
- Mice Lacking p27 Display Increased Body Size, Multiple Organ Hyperplasia, Retinal Dysplasia, and Pituitary TumorsCell, 1996
- Role of the Ubiquitin-Proteasome Pathway in Regulating Abundance of the Cyclin-Dependent Kinase Inhibitor p27Science, 1995
- Inhibitors of mammalian G1 cyclin-dependent kinases.Genes & Development, 1995
- Cyclic AMP-induced G1 phase arrest mediated by an inhibitor (p27Kip1) of cyclin-dependent kinase 4 activationCell, 1994
- Cloning of p27Kip1, a cyclin-dependent kinase inhibitor and a potential mediator of extracellular antimitogenic signalsCell, 1994
- p27Kip1, a cyclin-Cdk inhibitor, links transforming growth factor-beta and contact inhibition to cell cycle arrest.Genes & Development, 1994
- Formation and Activation of a Cyclin E-cdk2 Complex During the G 1 Phase of the Human Cell CycleScience, 1992