The Protooncogene Ski Controls Schwann Cell Proliferation and Myelination
Open Access
- 19 August 2004
- journal article
- research article
- Published by Elsevier
- Vol. 43 (4) , 499-511
- https://doi.org/10.1016/j.neuron.2004.08.001
Abstract
No abstract availableKeywords
This publication has 62 references indexed in Scilit:
- TGFβ1 Modulates the Phenotype of Schwann Cells at the Transcriptional LevelMolecular and Cellular Neuroscience, 2002
- Proliferation of Schwann cells and regulation of cyclin D1 expression in an animal model of Charcot‐Marie‐Tooth disease type 1AJournal of Neuroscience Research, 2002
- Differential Cyclin D1 Requirements of Proliferating Schwann Cells during Development and after InjuryMolecular and Cellular Neuroscience, 2001
- c-Ski Acts as a Transcriptional Co-repressor in Transforming Growth Factor-β Signaling through Interaction with SmadsJournal of Biological Chemistry, 1999
- MAG-deficient schwann cells myelinate dorsal root ganglion neurons in cultureGlia, 1998
- Mice lacking the ski proto-oncogene have defects in neurulation, craniofacial patterning, and skeletal muscle developmentGenes & Development, 1997
- N-Oct 5 is generated by in vitro proteolysis of the neural POU-domain protein N-Oct 3Oncogene, 1997
- Tst-1/Oct-6/SCIP regulates a unique step in peripheral myelination and is required for normal respiration.Genes & Development, 1996
- Hypermyelination and demyelinating peripheral neuropathy in Pmp22-deficient miceNature Genetics, 1995
- The ski oncogene induces muscle differentiation in quail embryo cellsCell, 1989