PPARγ Is Required for the Differentiation of Adipose Tissue In Vivo and In Vitro
- 1 October 1999
- journal article
- Published by Elsevier in Molecular Cell
- Vol. 4 (4) , 611-617
- https://doi.org/10.1016/s1097-2765(00)80211-7
Abstract
No abstract availableKeywords
Funding Information
- National Institutes of Health (KO8-DK02535, R01-HL58606, R37 DK-31405)
- American Heart Association
This publication has 24 references indexed in Scilit:
- Rat Preputial Sebocyte Differentiation Involves Peroxisome Proliferator-Activated Receptors11Preliminary reports were presented at IV International Dermatology Symposium, April 11 1997, Berlin; Biomedicine ‘97, April 25 1997, Washington, DC; and The Midwest Society for Pediatric Research, Chicago, September 25 1997.Journal of Investigative Dermatology, 1999
- PPARγ Promotes Monocyte/Macrophage Differentiation and Uptake of Oxidized LDLCell, 1998
- Oxidized LDL Regulates Macrophage Gene Expression through Ligand Activation of PPARγCell, 1998
- Terminal Differentiation of Human Breast Cancer through PPARγMolecular Cell, 1998
- 15-Deoxy-Δ12,14-Prostaglandin J2 is a ligand for the adipocyte determination factor PPARγCell, 1995
- Transdifferentiation of myoblasts by the adipogenic transcription factors PPAR gamma and C/EBP alpha.Proceedings of the National Academy of Sciences, 1995
- Cascade regulation of terminal adipocyte differentiation by three members of the C/EBP family of leucine zipper proteins.Genes & Development, 1995
- Stimulation of adipogenesis in fibroblasts by PPARγ2, a lipid-activated transcription factorCell, 1994
- mPPAR gamma 2: tissue-specific regulator of an adipocyte enhancer.Genes & Development, 1994
- Control of the peroxisomal β-oxidation pathway by a novel family of nuclear hormone receptorsPublished by Elsevier ,1992