Abstract
There is increasing evidence that the post‐prandial state is an important contributing factor to the development of atherosclerosis. In non‐diabetic subjects the atherosclerotic risk factors comprised in the categories of lipids, coagulation system and endothelial function may be adversely modified in the post‐prandial phase. The generation of an oxidative stress may be the common pathway through which eating may induce these alterations. In diabetic patients these phenomena may be amplified by post‐prandial hyperglycemia. Copyright © 2000 John Wiley & Sons, Ltd.