Protein Kinase B Stimulates the Translocation of GLUT4 but Not GLUT1 or Transferrin Receptors in 3T3-L1 Adipocytes by a Pathway Involving SNAP-23, Synaptobrevin-2, and/or Cellubrevin
Open Access
- 1 October 1999
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 274 (40) , 28087-28095
- https://doi.org/10.1074/jbc.274.40.28087
Abstract
No abstract availableKeywords
This publication has 60 references indexed in Scilit:
- Mechanism of activation and function of protein kinase BCurrent Opinion in Genetics & Development, 1998
- From receptor to transporter: insulin signalling to glucose transportDiabetologia, 1997
- The IRS‐signalling system during insulin and cytokine actionBioEssays, 1997
- Potential Role of Protein Kinase B in Glucose Transporter 4 Translocation in Adipocytes*Endocrinology, 1997
- Expression of a Constitutively Active Akt Ser/Thr Kinase in 3T3-L1 Adipocytes Stimulates Glucose Uptake and Glucose Transporter 4 TranslocationJournal of Biological Chemistry, 1996
- Overexpression of a Constitutively Active Form of Phosphatidylinositol 3-Kinase Is Sufficient to Promote Glut 4 Translocation in AdipocytesJournal of Biological Chemistry, 1996
- Activated Phosphatidylinositol 3-Kinase Is Sufficient to Mediate Actin Rearrangement and GLUT4 Translocation in 3T3-L1 AdipocytesPublished by Elsevier ,1996
- Compartmentalization of protein traffic in insulin-sensitive cellsAmerican Journal of Physiology-Endocrinology and Metabolism, 1996
- SNAREs and targeted membrane fusionFEBS Letters, 1995
- Inhibition of the translocation of GLUT1 and GLUT4 in 3T3-L1 cells by the phosphatidylinositol 3-kinase inhibitor, wortmanninBiochemical Journal, 1994