Mechanisms of postischemic contractile dysfunction
- 1 November 1999
- journal article
- review article
- Published by Elsevier in The Annals of Thoracic Surgery
- Vol. 68 (5) , 1898-1904
- https://doi.org/10.1016/s0003-4975(99)01035-8
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- Direct evidence that the hydroxyl radical plays a pathogenetic role in myocardial "stunning" in the conscious dog and demonstration that stunning can be markedly attenuated without subsequent adverse effects.Circulation Research, 1993
- Mechanism of myocardial "stunning".Circulation, 1990
- Is “stunned myocardium” a protective mechanism? Effect of acute recruitment and acute β-blockade on recovery of contractile function and high-energy phosphate stores at 1 day post-reperfusionAmerican Heart Journal, 1989
- Demonstration of free radical generation in "stunned" myocardium of intact dogs with the use of the spin trap alpha-phenyl N-tert-butyl nitrone.Journal of Clinical Investigation, 1988
- Evidence for a role of iron-catalyzed oxidants in functional and metabolic stunning in the canine heart.Circulation Research, 1988
- Attenuation of dysfunction in the postischemic 'stunned' myocardium by dimethylthiourea.Circulation, 1987
- Superoxide dismutase plus catalase improve contractile function in the canine model of the "stunned myocardium".Circulation Research, 1986
- Enhancement of recovery of myocardial function by oxygen free-radical scavengers after reversible regional ischemia.Circulation, 1985
- The stunned myocardium: prolonged, postischemic ventricular dysfunction.Circulation, 1982
- Regional myocardial functional and electrophysiological alterations after brief coronary artery occlusion in conscious dogs.Journal of Clinical Investigation, 1975