Wounding Smad
- 1 September 1999
- journal article
- Published by Springer Nature in Nature Cell Biology
- Vol. 1 (5) , E117-E119
- https://doi.org/10.1038/12944
Abstract
No abstract availableKeywords
This publication has 6 references indexed in Scilit:
- Mice lacking Smad3 show accelerated wound healing and an impaired local inflammatory responseNature Cell Biology, 1999
- Targeted Disruption of Smad3 Reveals an Essential Role in Transforming Growth Factor β-Mediated Signal TransductionMolecular and Cellular Biology, 1999
- Targeted disruption of SMAD3 results in impaired mucosal immunity and diminished T cell responsiveness to TGF-betaThe EMBO Journal, 1999
- Induction of apoptosis by Smad3 and down-regulation of Smad3 expression in response to TGF-β in human normal lung epithelial cellsOncogene, 1998
- Smad3 Mutant Mice Develop Metastatic Colorectal CancerCell, 1998
- TGF-β SIGNAL TRANSDUCTIONAnnual Review of Biochemistry, 1998