Targeting endothelial cells with adenovirus expressing nitric oxide synthase prevents elevation of blood pressure in stroke-prone spontaneously hypertensive rats
Open Access
- 1 August 2005
- journal article
- research article
- Published by Elsevier in Molecular Therapy
- Vol. 12 (2) , 321-327
- https://doi.org/10.1016/j.ymthe.2005.02.025
Abstract
No abstract availableKeywords
This publication has 43 references indexed in Scilit:
- Cardiovascular Disease in Non-Western CountriesNew England Journal of Medicine, 2004
- Endothelial FunctionCirculation, 2004
- Gene and cell‐based therapies for heart diseaseThe FASEB Journal, 2004
- Extracellular Superoxide Dismutase Is a Major Determinant of Nitric Oxide BioavailabilityCirculation Research, 2003
- A Critical Review of the Role of Endothelial Factors in the Pathogenesis of HypertensionJournal of Cardiovascular Pharmacology, 2001
- Endothelial Dysfunction in Cardiovascular Diseases: The Role of Oxidant StressCirculation Research, 2000
- Gene transfer of endothelial nitric oxide synthase but not Cu/Zn superoxide dismutase restores nitric oxide availability in the SHRSPCardiovascular Research, 2000
- Gene Transfer of Endothelial Nitric Oxide Synthase Reduces Angiotensin II–Induced Endothelial DysfunctionHypertension, 2000
- Inhibition of Late Vein Graft Neointima Formation in Human and Porcine Models by Adenovirus-Mediated Overexpression of Tissue Inhibitor of Metalloproteinase-3Circulation, 2000
- Gene transfer of endothelial nitric oxide synthase improves nitric oxide-dependent endothelial function in a hypertensive rat model.Cardiovascular Research, 1999