Flow Shear Stress Stimulates Gab1 Tyrosine Phosphorylation to Mediate Protein Kinase B and Endothelial Nitric-oxide Synthase Activation in Endothelial Cells

This publication has 42 references indexed in Scilit:

• The Docking Protein Gab1 Is an Essential Component of an Indirect Mechanism for Fibroblast Growth Factor Stimulation of the Phosphatidylinositol 3-Kinase/Akt Antiapoptotic Pathway
  Molecular and Cellular Biology, 2004
• VEGF signalling: integration and multi-tasking in endothelial cell biology
  Biochemical Society Transactions, 2003
• Ligand-Independent Activation of Vascular Endothelial Growth Factor Receptor 2 by Fluid Shear Stress Regulates Activation of Endothelial Nitric Oxide Synthase
  Circulation Research, 2003
• Signal transduction by vascular endothelial growth factor receptors
  Biochemical Society Transactions, 2003
• Evidence for a role of platelet endothelial cell adhesion molecule-1 in endothelial cell mechanosignal transduction
  The Journal of cell biology, 2002
• Receptor-Specific Regulation of Phosphatidylinositol 3′-Kinase Activation by the Protein Tyrosine Phosphatase Shp2
  Molecular and Cellular Biology, 2002
• The gift of Gab
  FEBS Letters, 2002
• Requirement of SHP2 Binding to Grb2-associated Binder-1 for Mitogen-activated Protein Kinase Activation in Response to Lysophosphatidic Acid and Epidermal Growth Factor
  Published by Elsevier ,2000
• Endothelial vasodilator dysfunction: Pathogenetic link to myocardial ischaemia or epiphenomenon?
  The Lancet, 1996
• A Grb2-associated docking protein in EGF- and insulin-receptor signalling
  Nature, 1996