Genetic dissection of susceptibility to radiation-induced apoptosis of thymocytes and mapping of Rapop1, a novel susceptibility gene
- 10 February 1995
- Vol. 25 (3) , 609-614
- https://doi.org/10.1016/0888-7543(95)80001-3
Abstract
No abstract availableKeywords
This publication has 32 references indexed in Scilit:
- Induction of apoptosis in fibroblasts by IL-1β-converting enzyme, a mammalian homolog of the C. elegans cell death gene ced-3Cell, 1993
- The C. elegans cell death gene ced-3 encodes a protein similar to mammalian interleukin-1β-converting enzymeCell, 1993
- Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programed cell deathCell, 1993
- bcl-x, a bcl-2-related gene that functions as a dominant regulator of apoptotic cell deathCell, 1993
- p53 is required for radiation-induced apoptosis in mouse thymocytesNature, 1993
- Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosisNature, 1992
- bcl-2 inhibits multiple forms of apoptosis but not negative selection in thymocytesCell, 1991
- Induction of bcl-2 expression by epstein-barr virus latent membrane protein 1 protects infected B cells from programmed cell deathCell, 1991
- Bcl-2 gene promotes haemopoietic cell survival and cooperates with c-myc to immortalize pre-B cellsNature, 1988
- Glucocorticoid-induced thymocyte apoptosis is associated with endogenous endonuclease activationNature, 1980