Alzheimer's disease: is there a problem beyond recognition?
- 31 May 1990
- journal article
- review article
- Published by Elsevier in Trends in Pharmacological Sciences
- Vol. 11 (5) , 183-184
- https://doi.org/10.1016/0165-6147(90)90110-t
Abstract
No abstract availableThis publication has 14 references indexed in Scilit:
- Disease states can modify both receptor number and signal transduction pathwaysTrends in Pharmacological Sciences, 1989
- Inositol phosphates and cell signallingNature, 1989
- Coupling of D1 dopamine receptors to the guanine nucleotide binding protein Gs is deficient in Huntington's diseaseBrain Research, 1989
- Receptors that inhibit phosphoinositide breakdownTrends in Pharmacological Sciences, 1989
- Receptor-mediated stimulation of inositol phospholipid hydrolysis in human brainEuropean Journal of Pharmacology, 1989
- Decreased brain [3H]inositol 1,4,5-trisphosphate binding in Alzheimer's diseaseNeuroscience Letters, 1988
- Decreased levels of protein kinase C in Alzheimer brainBrain Research, 1988
- VIP-sensitive adenylate cyclase, guanylate cyclase, muscarinic receptors, choline acetyltransferase and acetylcholinesterase, in brain tissue afflicted by alzheimer's disease/senile dementia of the Alzheimer typeNeurobiology of Aging, 1988
- Reduced Phosphoinositide Concentrations in Anterior Temporal Cortex of Alzheimer‐Diseased BrainsJournal of Neurochemistry, 1987
- Transmitter deficits in Alzheimer's diseaseNeurochemistry International, 1985