Split ends antagonizes the Notch and potentiates the EGFR signaling pathways during Drosophila eye development
- 31 October 2007
- journal article
- Published by Elsevier in Mechanisms of Development
- Vol. 124 (9-10) , 792-806
- https://doi.org/10.1016/j.mod.2007.05.002
Abstract
No abstract availableKeywords
This publication has 103 references indexed in Scilit:
- Rbm15 Modulates Notch-Induced Transcriptional Activation and Affects Myeloid DifferentiationMolecular and Cellular Biology, 2007
- Msx2-interacting nuclear target protein (Mint) deficiency reveals negative regulation of early thymocyte differentiation by Notch/RBP-J signalingProceedings of the National Academy of Sciences, 2007
- A family of Rhomboid intramembrane proteases activates all Drosophila membrane-tethered EGF ligandsThe EMBO Journal, 2002
- Notch Activation of yan Expression Is Antagonized by RTK/Pointed Signaling in the Drosophila EyeCurrent Biology, 2002
- Drosophila Rhomboid-1 Defines a Family of Putative Intramembrane Serine ProteasesCell, 2001
- Notch Signaling: Cell Fate Control and Signal Integration in DevelopmentScience, 1999
- Evolution of proneural atonal expression during distinct regulatory phases in the developing Drosophila eyeCurrent Biology, 1996
- Suppressor of hairless directly activates transcription of enhancer of split complex genes in response to Notch receptor activity.Genes & Development, 1995
- The neurogenic suppressor of hairless DNA-binding protein mediates the transcriptional activation of the enhancer of split complex genes triggered by Notch signaling.Genes & Development, 1995
- Developmental control by the Drosophila EGF receptor homolog DERTrends in Genetics, 1991