Modulation of mitochondrial K+ permeability and reactive oxygen species production by the p13 protein of human T-cell leukemia virus type 1
Open Access
- 11 February 2009
- journal article
- Published by Elsevier in Biochimica et Biophysica Acta (BBA) - Bioenergetics
- Vol. 1787 (7) , 947-954
- https://doi.org/10.1016/j.bbabio.2009.02.001
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- Superoxide Flashes in Single MitochondriaCell, 2008
- Recent progress in elucidating the molecular mechanism of the mitochondrial permeability transition poreBiochimica et Biophysica Acta (BBA) - Bioenergetics, 2008
- The mitochondrial permeability transition pore and its involvement in cell death and in disease pathogenesisApoptosis, 2007
- Mitochondrial potassium channels: From pharmacology to functionBiochimica et Biophysica Acta (BBA) - Bioenergetics, 2006
- Relevance of CREB phosphorylation in the anti-apoptotic function of human T-lymphotropic virus type 1 tax protein in serum-deprived murine fibroblastsExperimental Cell Research, 2004
- ViroporinsPublished by Wiley ,2003
- Mitochondrial Alterations Induced by the p13II Protein of Human T-cell Leukemia Virus Type 1Journal of Biological Chemistry, 2002
- Block of a Mitochondrial-Mediated Apoptotic Pathway in Tax-Expressing Murine FibroblastsExperimental Cell Research, 2001
- Opening of the Mitochondrial Permeability Transition Pore Causes Depletion of Mitochondrial and Cytosolic NAD+and Is a Causative Event in the Death of Myocytes in Postischemic Reperfusion of the HeartJournal of Biological Chemistry, 2001
- Solid phase peptide synthesis utilizing 9‐fluorenylmethoxycarbonyl amino acidsInternational Journal of Peptide and Protein Research, 1990