Nitric oxide antagonizes the actions of angiotensin II to enhance tubuloglomerular feedback responsiveness
- 1 November 1995
- journal article
- Published by Elsevier in Kidney International
- Vol. 48 (5) , 1406-1411
- https://doi.org/10.1038/ki.1995.429
Abstract
No abstract availableKeywords
This publication has 13 references indexed in Scilit:
- Reabsorption of nitro-L-arginine infused into the late proximal tubule participates in modulation of TGF responsivenessKidney International, 1995
- Recommendations for the health monitoring of mouse, rat, hamster, guineapig and rabbit breeding colonies: Report of the Federation of European Laboratory Animal Science Associations (FELASA) Working Group on Animal Health accepted by the FELASA Board of Management November 1992Laboratory Animals, 1994
- Evidence for the role of nitric oxide in macula densa control of glomerular hemodynamics.Journal of Clinical Investigation, 1993
- Resetting of the tubuloglomerular feedback mechanism by blockade of NO‐synthaseActa Physiologica Scandinavica, 1993
- Sodium and angiotensin in hypertension induced by long-term nitric oxide blockade.Hypertension, 1993
- Endothelium-derived relaxing factor/nitric oxide modulates angiotensin II action in the isolated microperfused rabbit afferent but not efferent arteriole.Journal of Clinical Investigation, 1993
- Nitric oxide synthase in macula densa regulates glomerular capillary pressure.Proceedings of the National Academy of Sciences, 1992
- Nitric oxide modulates vascular tone in preglomerular arterioles.Hypertension, 1992
- Modulation of angiotensin II-induced vasoconstriction by endothelium-derived relaxing factor in the isolated microperfused rabbit afferent arteriole.Journal of Clinical Investigation, 1991
- Endogenous angiotensin concentrations in specific intrarenal fluid compartments of the rat.Journal of Clinical Investigation, 1990