Rapid metabolic inactivation is the basis for cross-resistance to chlorsulfuron in diclofop-methyl-resistant rigid ryegrass (Lolium rigidum) biotype
- 1 July 1992
- journal article
- Published by Elsevier in Pesticide Biochemistry and Physiology
- Vol. 43 (3) , 182-192
- https://doi.org/10.1016/0048-3575(92)90032-u
Abstract
No abstract availableThis publication has 19 references indexed in Scilit:
- Atrazine Resistance in a Velvetleaf (Abutilon theophrasti) Biotype Due to Enhanced Glutathione S-Transferase ActivityPlant Physiology, 1991
- Metabolism of diclofop-methyl in susceptible and resistant biotypes of Lolium rigidumPesticide Biochemistry and Physiology, 1991
- Cross-Resistance to Herbicides in Annual Ryegrass (Lolium rigidum)Plant Physiology, 1990
- Herbicide-resistant Weeds in AustraliaWeed Technology, 1990
- Atrazine resistance in velvetleaf (Abutilon theophrasti) due to enhanced atrazine detoxificationPesticide Biochemistry and Physiology, 1989
- Fatty-acid biosynthesis and acetyl-coa carboxylase as a target of diclofop, fenoxaprop and other aryloxy-phenoxy-propionic acid herbicidesZeitschrift für Naturforschung C, 1988
- Inhibition of plant acetyl-coenzyme a carboxylase by the herbicides sethoxydim and haloxyfopBiochemical and Biophysical Research Communications, 1987
- The occurrence of herbicide cross-resistance in a population of annual ryegrass, Lolium rigidum, resistant to diclofop-methylAustralian Journal of Agricultural Research, 1986
- ImidazolinonesPlant Physiology, 1984
- Site of Action of ChlorsulfuronPlant Physiology, 1984