Abstract
Experimental evidence is now available demonstrating that the great majority of malignant tumors in animals, as well as the leukemias and lymphomas, are caused by transmissible oncogenic viruses. These viruses are widely disseminated in a variety of animal species and most probably in human beings; they are frugal and moderate in their requirements, causing no harm to their carrier hosts. However, when triggered by a variety of metabolic, hormonal or chemical factors or by ionizing radiation, they become pathogenic and cause the development of malignant tumors or leukemia in their hosts.1 Oncogenic viruses probably represent a very old, latent infection . . .