Sustained Phosphorylation of Bid Is a Marker for Resistance to Fas-Induced Apoptosis During Chronic Liver Diseases
Open Access
- 1 January 2006
- journal article
- Published by Elsevier in Gastroenterology
- Vol. 130 (1) , 104-119
- https://doi.org/10.1053/j.gastro.2005.10.012
Abstract
No abstract availableKeywords
This publication has 69 references indexed in Scilit:
- Suppression of Calcium Release from Inositol 1,4,5-Trisphosphate-sensitive Stores Mediates the Anti-apoptotic Function of Nuclear Factor-κBJournal of Biological Chemistry, 2005
- Oncosis represents the main type of cell death in mouse models of cholestasisJournal of Hepatology, 2004
- Regulation of α-Fetoprotein by Nuclear Factor-κB Protects Hepatocytes from Tumor Necrosis Factor-α Cytotoxicity during Fetal Liver Development and Hepatic OncogenesisCancer Research, 2004
- Reduced oncotic necrosis in Fas receptor-deficient C57BL/6J-lpr mice after bile duct ligationHepatology, 2004
- Reduced oncotic necrosis in fas receptor-deficient C57BL/6J- lpr mice after bile duct ligationHepatology, 2004
- NF-κB functions as a tumour promoter in inflammation-associated cancerNature, 2004
- Mechanism of Phosphorylation-Dependent Binding of APC to β-Catenin and Its Role in β-Catenin DegradationMolecular Cell, 2004
- The Bcl-2 family: roles in cell survival and oncogenesisOncogene, 2003
- Phosphorylation of calsenilin at Ser63 regulates its cleavage by caspase-3Molecular and Cellular Neuroscience, 2003
- Proapoptotic BAX and BAK: A Requisite Gateway to Mitochondrial Dysfunction and DeathScience, 2001