Involvement of Cyclooxygenase-2 in the Potentiation of Allyl Alcohol-Induced Liver Injury by Bacterial Lipopolysaccharide
- 1 July 2001
- journal article
- Published by Elsevier in Toxicology and Applied Pharmacology
- Vol. 174 (2) , 113-121
- https://doi.org/10.1006/taap.2001.9183
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- Bacterial Lipopolysaccharide Enhances Aflatoxin B1 Hepatotoxicity in Rats by A Mechanism That Depends on Tumor Necrosis Factor αHepatology, 2001
- Lipopolysaccharide Augments Aflatoxin B1-Induced Liver Injury through Neutrophil-Dependent and -Independent MechanismsToxicological Sciences, 2000
- Bacterial lipopolysaccharide exposure augments aflatoxin B(1)-induced liver injury.Toxicological Sciences, 2000
- Induction by interleukin 6 of Gs-coupled prostaglandin E2 receptors in rat hepatocytes mediating a prostaglandin E2-dependent inhibition of the hepatocyte’s acute phase responseHepatology, 2000
- Role of prostaglandins generated by cyclooxygenase-1 and cyclooxygenase-2 in healing of ischemia–reperfusion-induced gastric lesionsEuropean Journal of Pharmacology, 1999
- Differential expression of prostanoid receptors in hepatocytes, Kupffer cells, sinusoidal endothelial cells and stellate cells of rat liverJournal of Hepatology, 1999
- Structural specificity for prostaglandin effects on hepatocyte glycogenolysisBiochemical Journal, 1990
- Comparative Study of Cytotoxicity, Tumor Necrosis Factor, and Prostaglandin Release After Stimulation of Rat Kupffer Cells, Murine Kupffer Cells, and Murine Inflammatory Liver MacrophagesJournal of Leukocyte Biology, 1989
- Conditioned media of Kupffer and endothelial liver cells influence protein phosphorylation in parenchymal liver cells. Involvement of prostaglandinsBiochemical Journal, 1988
- Endotoxin induced hepatic necrosis in rats on an alcohol dietThe Journal of Pathology, 1987